Zinc is crucial for enzymatic reactions, immune modulation, and neuroprotection, supporting cell proliferation, collagen synthesis, and reducing oxidative stress. By stabilizing cellular antioxidants and inhibiting inflammatory markers, zinc aids in tissue repair and immune regulation. It supports neurotransmitter function, facilitating long-term potentiation essential for cognition. Zinc deficiency may impair immune function, wound healing, and cognitive processes, underscoring its significance in therapeutic protocols.

Mechanisms of Action and Benefits:

• Supports over 50 enzymes across all six enzyme classes
• Catalyzes protein synthesis and collagen formation
• Inhibits caspase enzymes, reducing apoptosis
• Enhances antioxidant defenses, inducing metallothioneins
• Mitigates inflammatory markers (e.g., C-reactive protein)
• Facilitates neurotransmitter function and LTP for memory
• Supports immune cell functions (macrophages, neutrophils, NK cells)
• Contributes to mucosal barrier integrity
• Aids in wound healing through epithelialization and infection resistance

References

1. https://lpi.oregonstate.edu/mic/minerals/zinc accessed October 2024

2. https://lpi.oregonstate.edu/mic/health-disease/cognitive-function#zinc-deficiency accessed October 2024

3. Stefano L. Sensi, Pierre Paoletti, Jae-Young Koh, Elias Aizenman, Ashley I. Bush and Michal Hershfinkel. Journal of Neuroscience 9 November 2011, 31 (45)